Silencers and suppressors aren't necessarily only for assassins. They are extremely useful for gun enthusiasts who go regularly to the range and for newbies. The advantage comes with no damage to your ears, and the ability to hear safety instructions. Without the suppressor, you can have permanent ear damage even with the ear muffs if you shoot regularly. Anyway, in this video notice the quietness of the MP5 and the automatic fire of the glock 18c! Amazing!

A funny compilation of some practice air drops of the US military that didn't go exactly as planed. They're very funny if you don't start to count the repair costs

Did you think it's easy to find a football partner? ;)

Wow. If you want to see something go bad, give a first timer a .50 caliber Desert Eagle to shoot, and then close your eyes. This is exactly what happens in this video. Ouch!

Actually, pretty slim. But the dude must've practiced a day or so, until he got it perfect. It's a very elaborate Halo 3 suicide. Wow!

This guy should have been born at least 150-200 years ago when knowing how to use a weapon like this would have made him a hero (or at least Jesse James). Hell...it doesn't get better than this guy. Enjoy his skills!

For the first time a gene was identified that allows the repair of damaged nerves in nematodes. The study is from Science Express. A gene that can stimulate the growth of nerve cells was first identified by researchers at the University of Utah (USA), thanks to cutting-edge experimental techniques and a huge genetic screening on a nematode (cylindrical or worm). The neurons, which in the embrio are able to regenerate, in adults have their capacity to "repair" reduced or absent. In other words, damage to the central nervous system (brain or spinal cord) and its consequences - paralysis, loss or reduction of cognitive faculties - are permanent. "In the past molecules have been identified that can inhibit the growth of neurons in different organisms," says the coordinator of research Michael Bastiani, "but their removal in the laboratory had no effect. That is why we went to look for those genes that can stimulate rather than inhibit, the regeneration of nerve. " Taking as a experimental model flat worms (Caenorhabditis elegans), biologists have searched for the genes that trigger the regrowth of motor (neurons that "command" voluntary muscles): in practice, with an experimental technique called RNA interference to "shut down ", one by one, 5000 on 20,000 genes in the DNA of worms (genes similar are also present in humans). The analysis led to the identification of dlk-1, which appears to play a key role in the regeneration of nerve tissue, and three other genes responsible for the formation of axons (parts of the neuron that conduct electrical signal). The researchers found that in nematodes, the gene dlk-1 not only triggers a chain of events known as "Map kinase" behind the growth of neurons, but also that their regeneration can be increased or decreased by stimulating the gene to produce amounts more or less high of the protein dlk-1.

The molecule slows the proliferation of tumor cells while giving the time needed to repair the damage to their DNA. The discovery, made by Italian researchers IEA, is published in Nature. The secret of immortality of cancer stem cells - those that feed it and cause relapses because they're immune to chemotherapy - was unveiled. Their strength is the p21 protein that slows the proliferation, giving them the time needed to repair damage to DNA. In practice, it is as if these cells were able to rejuvenate indefinitely: no age, and thus do not die. By blocking the production of p21, however, you can make them vulnerable and hit the tumor at the root. The research was conducted in the laboratories of the European Institute of Oncology (IFOM-IEO) in collaboration with the universities of Milan and Perugia, and was published this week in Nature. The cells age and die because they accumulate damage and mistakes borne of DNA during cell divisions. To understand why this does not happen in a cancerous stem cell, the researchers observed what happens to a staminale "normal" when you alter one of the genes (oncogenes) that cause cancer (in this case, the acute myeloid leukemia). The study revealed that oncogenes stimulate the activity of another gene, called p21, and thus the production of the corresponding protein, whose effect is to slow the proliferation. In essence, these cells have much more time to repair other damaged DNA, and remain young and active, immune to chemotherapy drugs because they recognize and affect only the cells in rapid proliferation.

The switch that turns off and on to command the superconducting property of the new device is a trivial electric field. In practice, what has been done by Andrea Ankle and colleagues at the University of Geneva in the first superconducting transistors. The operation, represents a milestone of applied physics and paves the way for the development of a new generation of microchips - and therefore computers - much faster than at present. To understand how and why the device is considered so promising it must be from another discovery, made last year by the same group of university research in Switzerland and published in Science. In one study, physicists have created a single crystal in which two metal oxides (strontium titanate and lanthanum aluminate) are separated. Between these two materials, researchers have found a layer of free electrons (electronic cloud) and 0.3 Kelvin - that is just above absolute zero - traveling without any resistance. At that temperature, the crystal becomes a superconductor. Scientists have now discovered how to turn off and turn on the superconductivity of this crystal at will, or modules, simply by applying an electric field to the point of contact between the two oxides. The result is a version of superconductive field effect transistors (FET) devices known in applied physics, able to switch from one state to a semiconductor insulator, and basic digital information in electronics (the fact that the current can pass or not is used as a binary 1-0 to store information). As the field effect transistors is a semiconductor, however, it always has resistance to the passage of current. This means that the speed at which you can get the electrons when the device is "on" is limited which means heat develops beyond a certain limit. This side effect is damaging the transistor. A superconducting transistor, however, can pass electrons (and record information) much more quickly, as it does not oppose any resistance to the passage of current and, therefore, not heat. There remains the problem of extremely low temperatures required for superconductivity. A limit that research is a long time trying to overcome.

The brain responds to stimuli, tactile and visual contradiction in delaying the processing of information that comes from the skin. A fly is laying on the right elbow. Slight itching, moving vision towards the elbow, identification of the intruder and its position and, finally, blow. This reaction is not as instantaneous as you can imagine. Indeed, the brain seems to delay affixed aware of the tactile perception, as reported a study in Current Biology Group for Research in Cognitive Neuroscience (Grnc) in Barcelona. The brain is often having to generate rapid responses integrating stimuli that produce information in contradiction: if, for example, the subject has crossed his arms and brings his right arm on the left side and left arm on the right side, his hands will be in a position that is reversed from the original location. In this case, the brain must be able to correctly integrate the information of the tactile stimulus (for example pinch) on the right hand, although the visual stimulus comes in fact from the left. To avoid mistakes the brain needs time to make a realignment of the information space of two different maps: that rof the body and one that covers everything else. To understand how the mechanism works, researchers have assessed the response of 32 university students to a series of visual and tactile stimuli. Each student has been subjected to 600 tests. "First we asked participants to cross the arms, so that the position of hands was in conflict with the anatomical position" says Salvador Soto-Faraco, one of the authors of the study, "we have stimulated one of two hands." Few tenths of a second later, a small light (visual stimulus) I had left or right. Both the tactile stimuli that those visual products were in a totally random. In addition, the flash of light could be generated 60 or 200 milliseconds after the tactile stimulus. In order to assess whether the time elapsed between the two stimuli does influence or not the answer.

A study explains how a yeast cell becomes cancerous: the fault is a chromosomal translocation An altering of the genome that causes cancer was finally detected and reproduced in the laboratory. The discovery, crucial for understanding the genesis and development of malignancies, is due to the geneticist Charles V. Bruschi, head of the Laboratory of Molecular Genetics of yeast, International Centre for Genetic Engineering and Biotechnology in Trieste (Icgeb) and coordinator of the Society of Italian scientific yeast (Zymi). Together with his group, Bruschi has uncovered, that the so-called chromosomal translocation is at fault. The yeast cells, whose DNA was sequenced completely in 1996, are a good model because they possess many similarities with mammalian cells and are easily manipulated by genetic engineering. Thanks to technical Bit (Bridge-Induced Translocation), designed by Bruschi and Valentina Tosato in 2005, it was possible to artificially induce the translocation and demonstrate the crucial role of this phenomenon in the formation of cancer. "Although it has long been a correlation between the presence of chromosomal translocations and the appearance of cancer cells," explains Bruschi, "so far it was not clear whether a translocation was the origin of cancer or whether, instead, it was a consequence. This is because we see patients when the cancer has already formed and in the cells already exists a particular translocation. In practice, these observations are made when it is too late to establish a relationship of cause and effect. "

A study analysis conducted over the past 27 years by British researchers, suggests that some Fans can reduce the risk of developing cancer. A review of studies published over the last 27 years suggests that the anti-inflammatory non-steroidal pharmacy drugs (Fans) such as aspirin could reduce the risk of breast cancer by as much as 20 per cent! In support, some experts of Guy's Hospital in London appeared in a publication of International Journal of Clinical Practice. According to Ian Fentiman, Fans could play an important role not only in prevention, but also as a therapy for women who have developed this type of cancer, combining their use with a hormonal treatment and using them as analgesics.