Here's a little something I like, and wanted to share with you all. A missile launch from a MLRS platform (Multiple Launch Rocket System). It's pretty spectacular, and probably doesn't compare to the actual feeling of being near it when it launches, but it's the best we can do. Enjoy!

Here's a cool "hi-tech" commercial from 1977 of the Sony Betamax system. It's cool to see how much we've gone forward in 30 years. Probably the best invention in this time was and is the Internet, and will be until someone finds a cure for cancer or AIDS. Until then, enjoy this 1977 Sony Betamax Commercial.

This is the 1st automated parking garage installed in New York. Fully automated, it functions similar to an elevator, by placing your car underground in a free parking spot. It's really cool considering that it saves not only your time, but also lots of space, and money because it doesn't have driveways!

Recipe: You take one portable RPL-300 Green Laser (around 316mW power output) and then you inflate 20 balloons or so until you start getting dizzy or bored (whichever comes first). You put the 20 balloons in a row, in front of the laser. The result is clearly visible in this video! P.S: As an alternative you can replace it with a several MW laser, add a tracking system to it, and call it an anti-rocket shield!

This very funny video features an innovative alarm design, which I guarantee you haven't heard off before. Hilarious! Enjoy!

Here's how a serious topic like the missile defense system can be condensed into 30 seconds of humorous info bulletin!

Here's the actual reason why we don't have autopilot landings on commercial jets. Lol. It didn't even try to abort

A guy, with lots of spare time on his hands decided to make a graffiti machine. It's actually similar to a plotter printer. It can draw by a system of pulleys which coordonated by the computer make the graffiti machine draw each point of the original image on the wall. Nice.

Imagine having a picture and being able to transform it into a 3D environment, and then navigating through it like you were there. What is more spectacular, is that this system would be able to transform the pictures from Google Earth into spectacular 3D environments. Wow.

With today's safety systems, car crashes that in the past would seem incredible to get out of, now result only in minor injuries. Cars are meant to break apart, just like the one in this video, so they can absorb much of the impact. Anyway, it's a spectacular crash!

This has to be the coolest robot I've seen this year (even cooler than Plen). It seems to work by using some suction system in its legs, and always holding 4 legs on the wall. So enjoy this cool video of the amazing climbing robot.

This is the Skyguard Laser Defense system, which is part of the missile defense system, able to track, and fire upon incoming air threats. In this video clip the Skyguard Laser Defense System is tested by destroying mortars, artillery fire and missiles launched from the ground. The Skyguard Laser Defense system is definitely the future in terms of the missile defense system which America is believed to have up-and-running.

This technology is bound to make it into safer systems against car bombs! As a demonstration one traffic pole, and one truck full of sandbags, go head to head. Only one will make it out alive. Who will it be? Just watch!

This is NASA WorldWind 1.4 and it makes Google Earth look like child's play. It just takes it to a next level. Besides exploring Earth, you can explore the Solar System's planets just like in Google Earth and see the volcanoes, craters and planet rings. It's plain awesome!

Can we act on stopping the process of infection, without the risk to develop strains resistant to antibiotics ? Small molecules that interrupt the chemical signals by which bacteria communicates by blocking the process of infection have been identified. The discovery, published in Molecular Cell, as well as representing a new option in the treatment of infections, reduces the risk of growth of bacteria strains resistant to antibiotics. Bacteria will exchange information with a system of intercellular communication, called quorum sensing, which allows them to perceive and respond to changes in density and to coordinate actions of the group. As soon as the conditions are favorable to population growth, for example if they are within a host, the bacteria sends chemical signals to molecules that bind to receptors inside: LuxR-type proteins or proteins of the type LuxN, located on membrane of each cell. In this way the infection proceeds without hitches. " Blocking the communications of the enemy has always been a winning weapon. The researchers searched the key to succeeding, and found in an old acquaintance. In a previous study Bassler and colleagues had discovered that a class of molecules called lactose acilomoserina (AHL), is able to compete with the signals acting on LuxN proteins, preventing them from binding to the receptor. In the recent study, researchers have realized that the AHL can also bind to proteins of the LuxR type. In this way was brought into light the AHL the ability to bind to both receptors, although the two proteins have two completely different structure and location mechanisms.

Italian researcher Alessandra Luchini wins the first edition of "The Prize Award” with a paper of a system to identify those molecules that signal the presence of a tumor (tumor markers) that are beyond the traditional methods of investigation. To do this requires making a hydrogel containing certain microscopic nano-spheres that once inserted in the samples of blood taken for analysis diagnostic trap some markers and protect them from deterioration. "These nano-spheres, made of the same plastic as hydrated soft contact lenses are equipped with special molecules that, once in the blood, snap-specific tumor markers and incorporate them. In this way, they protect them from enzymes that would otherwise deteriorate them. Usually blood tests fail to identify precisely because these markers are destroyed prematurely, " says researcher Alessandra Luchini. "The beauty of this system," says the researcher, "is that it does not need very sophisticated tools, which is simple and economical: with one hundred U.S. dollars we can make nano-spheres for more than two hundred patients." The new method is not going to replace the standard, but acts at a stage prior to analysis by providing a better quality.

Twenty years after the first partially successful attempt to cold fusion, a new experiment seems to have reopened the hopes of obtaining nuclear reactions at low energy (LENR low-energy nuclear reactions). This was announced by a team of researchers led by Pamela Mosier-Boss of the Space and Naval Warfare Systems Center San Diego (California), with a study presented at the annual meeting of American Chemical Society, the first visible evidence of the production of neutrons, the particles subatomic whose presence demonstrates the atomic reaction occurred. It was 1989 when Martin Fleischmann and Stanley Pons showed that it has obtained experimentally the Cold Fusion, arousing great outcry in the scientific community. Fusion is the reaction that takes place inside of stars, their source of energy, able to reproduce in the laboratory at room temperature this process would be an amazing achievement. Further research then disappointed initial expectations: the rare attempts (for example, those of 2000 and 2002) to reproduce the results of 1989 and have not convinced the path of nuclear reaction at low energy has not proved viable as an alternative to "clean" nuclear fission, which is based on the common operation of nuclear power.

The new devices can operate at 30 degrees above zero, rather than less than 70. This is the characteristic of the new generation of semiconductors, researched at the Italian Institute for the Physics of Matter (INFM-CNR), and in the Ludwig Maximilian University in Monaco of Bavaria and the ETH Zurich (the study). Today there are two ways to record information on a medium: the electronic format, in which the binary language is the passage of electrons (the transistors) and magnetic (MRAM memory), more recently, in which the binary language is given by state of magnetization. To communicate these two systems could boost significantly the computational schemes, pending the distant quantum computer. Doubling the processing power and memory of a chip while maintaining the size, without the need to go in nano-scale (a scale, that is, a billionth of a meter) are just two of the technology that promises magnetic semiconductors suggest a near future. These devices were made over ten years ago, but so far required temperatures far below zero to work. The problem now seems outdated as the known semiconductors gallium arsenide containing traces of manganese, a metal which has ferromagnetic properties at around 200 degrees below zero. To increase the temperature threshold, above which the ferromagnetic behavior disappears, the researchers deposited on a semiconductor film of iron - metal known for its magnetic properties - the thickness of a few nanometers. Iron and manganese interacted so effectively that the new material, has a ferromagnetic behavior up to 30 degrees above zero, a jump of over a hundred degrees above the starting temperature. This result is a technological response parallel to that of the race to miniaturization and the research was selected the American Physical Society as one of the most important published in Physical Review Letters

For the first time a gene was identified that allows the repair of damaged nerves in nematodes. The study is from Science Express. A gene that can stimulate the growth of nerve cells was first identified by researchers at the University of Utah (USA), thanks to cutting-edge experimental techniques and a huge genetic screening on a nematode (cylindrical or worm). The neurons, which in the embrio are able to regenerate, in adults have their capacity to "repair" reduced or absent. In other words, damage to the central nervous system (brain or spinal cord) and its consequences - paralysis, loss or reduction of cognitive faculties - are permanent. "In the past molecules have been identified that can inhibit the growth of neurons in different organisms," says the coordinator of research Michael Bastiani, "but their removal in the laboratory had no effect. That is why we went to look for those genes that can stimulate rather than inhibit, the regeneration of nerve. " Taking as a experimental model flat worms (Caenorhabditis elegans), biologists have searched for the genes that trigger the regrowth of motor (neurons that "command" voluntary muscles): in practice, with an experimental technique called RNA interference to "shut down ", one by one, 5000 on 20,000 genes in the DNA of worms (genes similar are also present in humans). The analysis led to the identification of dlk-1, which appears to play a key role in the regeneration of nerve tissue, and three other genes responsible for the formation of axons (parts of the neuron that conduct electrical signal). The researchers found that in nematodes, the gene dlk-1 not only triggers a chain of events known as "Map kinase" behind the growth of neurons, but also that their regeneration can be increased or decreased by stimulating the gene to produce amounts more or less high of the protein dlk-1.

The molecule slows the proliferation of tumor cells while giving the time needed to repair the damage to their DNA. The discovery, made by Italian researchers IEA, is published in Nature. The secret of immortality of cancer stem cells - those that feed it and cause relapses because they're immune to chemotherapy - was unveiled. Their strength is the p21 protein that slows the proliferation, giving them the time needed to repair damage to DNA. In practice, it is as if these cells were able to rejuvenate indefinitely: no age, and thus do not die. By blocking the production of p21, however, you can make them vulnerable and hit the tumor at the root. The research was conducted in the laboratories of the European Institute of Oncology (IFOM-IEO) in collaboration with the universities of Milan and Perugia, and was published this week in Nature. The cells age and die because they accumulate damage and mistakes borne of DNA during cell divisions. To understand why this does not happen in a cancerous stem cell, the researchers observed what happens to a staminale "normal" when you alter one of the genes (oncogenes) that cause cancer (in this case, the acute myeloid leukemia). The study revealed that oncogenes stimulate the activity of another gene, called p21, and thus the production of the corresponding protein, whose effect is to slow the proliferation. In essence, these cells have much more time to repair other damaged DNA, and remain young and active, immune to chemotherapy drugs because they recognize and affect only the cells in rapid proliferation.