Awesome Worms Armageddon skills shown to us in this video, especially with the Ninja Rope. Even if you've never played the game, I'm sure you'll be able to appreciate the stunts this guy can pull off with nothing more then some rope and lots of experience. Enjoy!

Can we act on stopping the process of infection, without the risk to develop strains resistant to antibiotics ? Small molecules that interrupt the chemical signals by which bacteria communicates by blocking the process of infection have been identified. The discovery, published in Molecular Cell, as well as representing a new option in the treatment of infections, reduces the risk of growth of bacteria strains resistant to antibiotics. Bacteria will exchange information with a system of intercellular communication, called quorum sensing, which allows them to perceive and respond to changes in density and to coordinate actions of the group. As soon as the conditions are favorable to population growth, for example if they are within a host, the bacteria sends chemical signals to molecules that bind to receptors inside: LuxR-type proteins or proteins of the type LuxN, located on membrane of each cell. In this way the infection proceeds without hitches. " Blocking the communications of the enemy has always been a winning weapon. The researchers searched the key to succeeding, and found in an old acquaintance. In a previous study Bassler and colleagues had discovered that a class of molecules called lactose acilomoserina (AHL), is able to compete with the signals acting on LuxN proteins, preventing them from binding to the receptor. In the recent study, researchers have realized that the AHL can also bind to proteins of the LuxR type. In this way was brought into light the AHL the ability to bind to both receptors, although the two proteins have two completely different structure and location mechanisms.

For the first time a gene was identified that allows the repair of damaged nerves in nematodes. The study is from Science Express. A gene that can stimulate the growth of nerve cells was first identified by researchers at the University of Utah (USA), thanks to cutting-edge experimental techniques and a huge genetic screening on a nematode (cylindrical or worm). The neurons, which in the embrio are able to regenerate, in adults have their capacity to "repair" reduced or absent. In other words, damage to the central nervous system (brain or spinal cord) and its consequences - paralysis, loss or reduction of cognitive faculties - are permanent. "In the past molecules have been identified that can inhibit the growth of neurons in different organisms," says the coordinator of research Michael Bastiani, "but their removal in the laboratory had no effect. That is why we went to look for those genes that can stimulate rather than inhibit, the regeneration of nerve. " Taking as a experimental model flat worms (Caenorhabditis elegans), biologists have searched for the genes that trigger the regrowth of motor (neurons that "command" voluntary muscles): in practice, with an experimental technique called RNA interference to "shut down ", one by one, 5000 on 20,000 genes in the DNA of worms (genes similar are also present in humans). The analysis led to the identification of dlk-1, which appears to play a key role in the regeneration of nerve tissue, and three other genes responsible for the formation of axons (parts of the neuron that conduct electrical signal). The researchers found that in nematodes, the gene dlk-1 not only triggers a chain of events known as "Map kinase" behind the growth of neurons, but also that their regeneration can be increased or decreased by stimulating the gene to produce amounts more or less high of the protein dlk-1.